Article
Short Communications

Sangram Biradar*, Muddsir Indikar**

*Associate Professor,

**Post-Graduate,

Department of Medicine, Basaveshwar Teaching & General Hospital, Kalaburagi.

Corresponding Author:

Dr Sangram Biradar, Associate Professor: Department of Medicine Basaveshwar Hospital, Kalaburagi 585105 drsangramb@yahoo.com.

Received Date: 2019-06-02,
Accepted Date: 2019-07-03,
Published Date: 2019-07-31
Year: 2019, Volume: 9, Issue: 3, Page no. 112, DOI: 10.26463/rjms.9_3_8
Views: 656, Downloads: 1
Licensing Information:
CC BY NC 4.0 ICON
This work is licensed under a Creative Commons Attribution-NonCommercial 4.0.
Abstract

None

<p>None</p>
Keywords
None
Downloads
  • 1
    FullTextPDF
Article

A 60 year male presented with a history of global aphasia and weakness of right upper and lower limb since 2 months with an insidious in onset and was non- progressive. The patient was a known case of hypertension since 5 years on irregular treatment. Investigation with 3D TOF M R Angiography showed complete occlusion of all C2 TO C6 (Petrous, lacerum, cavernous, and supraclinoid segments of left internal carotid artery, and koderate steno-occlusion of left middle cerebral artery (Image 1-3). MRI Brain showed left subacute infarct in left ganglio-capsular region, left corona radiata, left frontal and posterior parietal region and sub cortical white matter (Image 4), Vitamin B12 level showed< 50 pg/ml (Normal 120-180 pg/ml). Serum homocysteine levels were 32.7 micromol/l (5-13micromol/l) Irregular treatment for hypertension must have de-arranged vitamin B-12 levels and serum homocystein levels. It should be mentioned that high homocysteine levels has led to low vitamin B 12 levels. These are major risk factors for vascular disease.

Supporting File
References

None

We use and utilize cookies and other similar technologies necessary to understand, optimize, and improve visitor's experience in our site. By continuing to use our site you agree to our Cookies, Privacy and Terms of Use Policies.