Article
Review Article

Anagha D N1

1 : Associate Professor, Department of Roganidana and Vikriti vignana, Sushrutha Ayurvedic Medical College and Hospital, Jigani, Bengaluru.

Address for correspondence:

Anagha D N

Email: anagha.dn@gmail.com

Received Date: 2020-06-16,
Accepted Date: 2020-07-20,
Published Date: 2020-07-30
Year: 2020, Volume: 7, Issue: 2, Page no. 46-51, DOI: 10.26715/rjas.7_2_9
Views: 822, Downloads: 14
Licensing Information:
CC BY NC 4.0 ICON
This work is licensed under a Creative Commons Attribution-NonCommercial 4.0.
Abstract

Manas forms an important abode for manifestation of vikaras. It is acted upon by manasa as well as shareerika doshas. Affliction with imbalanced doshas and pathology in manovaha srotas contribute to the manifestation of manasa vikaras. A category of manasa vikaras, presenting with abnormal alterations with respect to motivational drive and the emotional outcome, is produced by vikrita sadhaka pitta. Shabda chikitsa and Geetam (Music) are the unique interventions framed in Ayurveda to manage manasavikaras, where sadhaka pitta is found to be a contributory cause. Hence a retrospective attempt is made in this article to prepare a review which gives scope for further research to establish the role of Music (Geetam) in managing psychiatric disorders

<p>Manas forms an important abode for manifestation of vikaras. It is acted upon by manasa as well as shareerika doshas. Affliction with imbalanced doshas and pathology in manovaha srotas contribute to the manifestation of manasa vikaras. A category of manasa vikaras, presenting with abnormal alterations with respect to motivational drive and the emotional outcome, is produced by vikrita sadhaka pitta. Shabda chikitsa and Geetam (Music) are the unique interventions framed in Ayurveda to manage manasavikaras, where sadhaka pitta is found to be a contributory cause. Hence a retrospective attempt is made in this article to prepare a review which gives scope for further research to establish the role of Music (Geetam) in managing psychiatric disorders</p>
Keywords
Sadhakapitta, Sadhakapittakrita Manasavikaras, Geetam, Music
Downloads
  • 1
    FullTextPDF
Article

INTRODUCTION

Manas in the context of disease is referred as satwa in Ayurveda. Manasavikaras develop in the background of involvement of rajas and tamas , affecting either manas alone or manas with shareera. The role of manovahasrotas has been identified in the pathology of manasavikaras. The vatadidoshas affect manas when it gets deprived of satwaguna and gets afflicted with rajo and tamogunas. The concept of mental health is identified as mere absence of Manasarogas.

SADHAKA PITTA

Hridaya is the sthana of sadhaka pitta. Accomplishment of aspirations (manoratha) by means of buddhi, medha, abhimana (ahankara) is an important function of sadhaka pitta . Aspirations are addressed as the purusharthas, which are directed in the form of Dharma, Artha, Kama and Moksha. Bahyagrahana and smarana are the most important phenomenon which mediates for the achievement of aspirations through sadhaka pitta. Another major function involves manifestation of states like shourya, bhaya, krodha, harsha, moha, prasadaetc. Hence the functions of sadhaka pitta can be summarized as abhiprarthita manoratha sadhana, bahyagrahana, smarana and shouryadi karana which are analysed as follows.

Abhiprarthita manoratha sadhana (Purushartha sadhana)

Purusharthas are the main objectives of life as explained in the science of Ayurveda. They give us a direction for our life and act as the motivational drive for the survival. They form a combination of conditioned behavior, homeostatic drive and survival instinct which formulate the abhiprarthita manorathas. The basic force required to achieve these aspirations is none other than sadhaka pitta, which functions through two important mechanisms i.e. Iccha and dwesha. Iccha is the force of desire and dwesha is the force of aversion which forms the potential for the manifestation of manasavikaras. Thus Iccha and dwesha can be understood for Reward and punishment systems of the brain respectively. Hence the entity of sadhaka pitta can be understood for the neurotransmitters associated with the functioning of Reward and the Punishment centers of the brain.

The reward system is a collection of brain structures and neural pathways that are responsible for reward-related cognition, including associative learning (primarily classical conditioning and operant reinforcement), incentive salience (i.e., motivation and desire), and positively-valence emotions, particularly emotions that involve pleasure. (e.g., joy, euphoria and ecstasy). The brain structures that compose the reward system are located primarily within the cortico-basal gangliathalamo-cortical loop. The basal ganglia portion of the loop drives activity within the reward system. Most of the pathways that connect structures within the reward system are glutamatergicinterneurons, GABAergicmedium spiny neurons (MSNs), and dopaminergicprojection neurons. Dopamine is responsible for regulation of motor behavior, pleasure related to motivation and also emotional arousal. It plays a critical role in the reward system. The reward and punishment centers are responsible for the behavioral and emotional changes in human beings. They are concerned with the affective nature of sensations i.e. reward (satisfaction/pleasant) and punishment (aversion/painful). Hence Indulgence and avoidance behavior is dependent on the functions of these centers. Attainment of purusharthas (motives) through reward (Iccha) and punish (dwesha) pathways occurs with the mediation of two important cognitive processes i.e. bahyagrahana and smarana. Hence reward and punish related cognition is an important function of sadhaka pitta which involves Incentive & Aversive salience (Bahyagrahana) and associative learning in the form of classical conditioning and operant reinforcement (Smarana).

Bahyagrahana (Motivational salience)

Sadhaka pitta causes accomplishment of motivational desires (abhishtita manorathas) through approach and avoidance behaviors towards the desires. This functional component can be considered for bahyagrahana. Motivational salience is a cognitive process and a form of attention that motivates or propels an individual's behavior towards or away from a particular object, perceived event or outcome. Motivational salience regulates the intensity of behaviors that facilitate the attainment of a particular goal, the amount of time and energy that an individual is willing to expend to attain a particular goal, and the amount of risk that an individual is willing to accept while working to attain a particular goal. Motivational salience is composed of two component processes that are defined by their attractive or aversive effects on an individual's behavior relative to a particular stimulus which include incentive salience and aversive salience. Incentive salience is the attractive form of motivational salience that causes approach behavior, and is associated with operant reinforcement, desirable outcomes, and pleasurable stimuli. Aversive salience is the aversive form of motivational salience that causes avoidance behavior, and is associated with operant punishment, undesirable outcomes, and unpleasant stimuli

Smarana (Learning and Conditioning)

Another important cognitive function of sadhaka pitta in achieving motives is smarana which can be understood for various conditioned reflexes which depend on previous experience. Rewarding stimuli can drive learning in both the form of classical conditioning (Pavlovian conditioning) and operant conditioning (instrumental conditioning). In classical conditioning, a reward can act as an unconditioned stimulus that, when associated with the conditioned stimulus, causes the conditioned stimulus to elicit both musculoskeletal (in the form of simple approach and avoidance behaviors) and vegetative responses. In operant conditioning, a reward may act as a reinforcer in that it increases or supports actions that lead to it.

Shauryadi karana (Valence)

Another important function of Sadhaka pitta is the emotional behavior in the form of shauryadi bhavas which represent positive and negative valence. Positive valence can be considered as the outcome of Iccha whereas negative valence can be considered as the outcome of dwesha. Valence or hedonic tone, is the affective quality referring to the intrinsic attractiveness or goodness (positive valence) or averseness or badness (negative valence) of an event, object, or situation.Hence the hedonic tone is responsible for feelings, affect, certain behaviors of approach and avoidance, goal attainment or nonattainment, and conformity with or violation of norms. Emotions of anger and fear have negative valence. Joy has positive valence.

Sadhakapittakrita Manasavikaras

The manasa vikaras categorized as Iccha and Dwesha based come under the perview of sadhaka pitta vikaras.Thus, Reward system disorders are Iccha based manasa vikaras and Punishment system disorders are Dwesha based manasa vikaras. The following table depicts sadhaka pittakrita manasa vikaras. 

Sadhaka pitta in akupitavastha (normal state) leads to shubha (equilibrium) of hridaya and manovaha srotas through normal states of shouryadi bhavas. In kupithavastha (pathological state) it leads to ashubha (disorder) in hridaya and manovaha srotas. Thus iccha and dwesha based manasavikaras will develop in the presence of pittaprakopaka and manasavikarakara nidana along with hridaya and manovahasroto dusti. Hence sadhakapittakrita manasa vikaras can be depicted for the psychiatric conditions associated with abnormal states of motivational salience. Lack of motivation, Anhedonia, Addiction, Paranoia are few conditions which can be considered for the category of above said manasavikaras.

Lack of motivation or drive is associated with dysfunctional motivational salience whereas Anhedonia (reduced capacity to feel pleasure) reflects blunted incentive salience. On the other end of the spectrum, heightened incentive salience that is narrowed for specific stimuli is characteristic of behavioral and drug addictions. Addiction is a brain disorder characterized by compulsive engagement in rewarding stimuli despite adverse consequences. The two properties that characterize all addictive stimuli are that they are reinforcing (i.e., they increase the likelihood that a person will seek repeated exposure to them) and intrinsically rewarding (i.e., they are perceived as being inherently positive, desirable, and pleasurable). In the case of fear or paranoia, dysfunction is associated with elevated aversive salience.

Shabda Chikitsa:

An Acess To Manas Shabda is an Indriyartha of Shrothrendriya. Manas has a significant role in controlling Indriyas. Hence in manasavikaras the control of manas is possible by utilizing shabda as a manipulative technique. The administered shabda for intervention should bring down the distress caused by these illnesses in the patient.

Geetam: A Pittadoshopakrama

Music is termed as Geetam (Ganam) in the literature of Ayurveda. Vocal or Instrumental mode of application of music is advised. Listening to music has been highlighted. Soothening music compatible to a person has to be selected. Geetam is a form of Adravya chikitsa. Adravya chikitsa is the therapeutic application of various biological principles or phenomenon in the form of noninvasive techniques. The clinical utility of music is implied to conditions caused due to afflicted Pitta dosha. Music can be employed as a main therapy or a regimen complimenting other treatment methods. Music Therapy

Music therapy

is an interpersonal process in which the therapist uses music and all its facets - physical, emotional, mental, social, spiritual and aesthetic - to help the client better, recover or maintain health.

Research On Music

Functional neuroimaging studies on music and emotion show that music can modulate activity in brain structures that are known to be crucially involved in emotion, such as the amygdala, nucleus accumbens, hypothalamus, hippocampus, insula, cingulate cortex and orbitofrontal cortex. Exposure to music also significantly increased serum calcium levels and neostriatal Dopamine levels thus causing a reduction in blood pressure. Auditory stimuli in the form of exposure to melodic music, increases Dopamine levels and the release of Serotonin in the Caudate - putamen, Nucleus accumbens & the areas linked to reward system in Rat forebrain suggesting that the music had a direct impact on monoamine activity in these brain areas.

DISCUSSION

The domain of influence of Geetam is over pitta dosha. Pitta exerts its functions over body as well as manas. Music corrects the lakshanas of Indriyadourbalyata and Alpanidrata produced as a result of pitta vitiation in the body. The vikrita pitta lakshanas of krodha, moha are resolved by music. Sadhaka pitta functions are updated in the form of upgrading the functions of buddhi and medha. Thus, music stabilizes the neurotransmitters responsible for the production of dysfunctional reward and punishment neural pathways. Music being a therapeutic sound acts on manovaha srotas. In manasavikaras, this is a unique approach of utilizing shabda as an intervention method. The principles of Harshana and Vismarana can be delivered through music therapy. Harshana implies to de-stressing the patient through release of specific neurotransmitters like dopamine. When reward areas are stimulated parasympathetic nerves are stimulated causing relaxation of body and mind. Sadhakapitta afflicted disorders are related to conditioned reflex related to motivational salience. Vismarana leads to changing or decoding the conditioned reflexes and their reinforcement which have led to dysfunctional motivational salience.

CONCLUSION

Sadhakapittakrita manasavikaras develop due to pathophysiological changes with respect to bahyagrahana and smarana of sadhaka pitta functions which manifest in terms of dysfunctional motivational salience. Music is a upakrama for pittapradhana vikaras and shabda chikitsa is a unique intervention for manasa vikaras. Thus, music can be utilized to manage manasa vikaras of sadhakapitta origin.

Supporting File
No Pictures
References

1. Vaidya Jadavaji Trikamji Acharya, Chakrapanidatta, Agnivesha. Charaka Samhita. Varanasi: Chaukhambha Prakashan; 2011. Chapter 1, Sootra Sthana, shloka. 55; p.15.

2. Vaidya Jadavaji Trikamji Acharya, Chakrapanidatta, Agnivesha. Ayurveda Deepika, Charaka Samhita. Varanasi: Chaukhambha Prakashan; 2011. Chapter 6, Vimana Sthana, shloka.5; p 254.

3. Vaidya JadavajiTrikamji Acharya, Susruta. Susruta samhita. Varanasi: Chaukhambha Sanskrit sansthan; 2009. Chapter 1, Sootra Sthana, shloka.26; p.7.

4. Vaidya Jadavaji Trikamji Acharya, Agnivesha. Charaka Samhita.Varanasi: Chaukhambha Prakashan; 2011. Chapter 9, Chikitsa Sthana, shloka.5; p.468.

5. Vaidya Jadavaji Trikamji Acharya, Agnivesha. Charaka Samhita.Varanasi: Chaukhambha Prakashan; 2011. Chapter 10, Chikitsa Sthana, shloka.58; p.477.

6. Priyavrat sharma, Gangadhara, Agnivesha. Caraka Samhita Critical Notes.Varanasi: 7th ed. Chaukambha Orientalia;2003. Vol 3.

7. Vaidya Jadavaji Trikamji Acharya, Chakrapanidatta, Agnivesha. Charaka Samhita. Varanasi: Chaukhambha Prakashan; 2011. Chapter 1, Sootra Sthana, shloka.55; p.15.

8. Pt Hari Sadashiva Sastri paradakara,Vagbhata. Astanga Hridaya. Varanasi: Chaukhambha Sanskrit samsthan; 2010. Chapter 12, Sutra sthana, shloka.13; p.194. Pt Hari Sadashiva Sastri paradakara, Arunadatta, Vagbhata. Sarvanga sundara, Astanga Hridaya. Varanasi: Chaukhambha Sanskrit samsthan; 2010. Chapter 1, Sutra sthana, shloka.20; p.14.

9. Vaidya Jadavaji Trikamji Acharya, Dalhana, Susruta. Nibandha sangraha,Susruta samhita. Varanasi: Chaukhambha Sanskrit sansthan; 2009. Chapter 21, Sootra Sthana, shloka.10; p.101.

10. Pt Hari Sadashiva Sastri paradakara, Arunadatta, Vagbhata. Sarvanga sundara, Astanga Hridaya. Varanasi: Chaukhambha Sanskrit samsthan; 2010. Chapter 12, Sutra sthana, shloka.13; p.194.

11. Pt Hari Sadashiva Sastri paradakara, Arunadatta, Vagbhata. Sarvanga sundara, Astanga Hridaya. Varanasi: Chaukhambha Sanskrit samsthan; 2010. Chapter 12, Sutra sthana, shloka 13; p.194.

12. Vaidya Jadavaji Trikamji Acharya, Chakrapanidatta, Susruta. Bhanumati, Susruta samhita. Varanasi: Chaukhambha Sanskrit sansthan; 2009. Chapter 21, Sootra Sthana, shloka.10; p.160.

13. Vaidya Jadavaji Trikamji Acharya, Dalhana, Susruta. Nibandha sangraha, Susruta samhita. Varanasi: Chaukhambha Sanskrit sansthan; 2009. Chapter 1, Sootra Sthana, shloka.33; p.6.

14. Schultz W (2015). "Neuronal reward and decision signals: from theories to data". 853– 951. doi:10.1152/physrev.00023.2014. PMC 4491543. PMID 26109341.

15. Yager LM, Garcia AF, Wunsch AM, Ferguson SM (August 2015). "The ins and outs of the striatum: Role in drug addiction". Neuroscience. 301: 529–541. doi:10.1016/j.neuroscience.2015.06.033. PMC 4523218. PMID 26116518.

16. Schacter, Gilbert and Weger. Psychology.United States of America. 2009. Print.

17. Schultz W (2015). "Neuronal reward and decision signals: from theories to data". 853–951. doi:10.1152/physrev.00023.2014. PMC 4491543. PMID 26109341.

18. Puglisi-Allegra S, Ventura R (June 2012). "Prefrontal/accumbal catecholamine system processes high motivational salience". Front. Behav. Neurosci. 6: 31. doi:10.3389/fnbeh.2012.00031. PMC 3384081. PMID 22754514. M

19. Schultz W (2015). "Neuronal reward and decision signals: from theories to data". 853–951. doi:10.1152/physrev.00023.2014. PMC 4491543. PMID 26109341

20. Koob GF, Moal ML (2006). Neurobiology of Addiction. Amsterdam: Elsevier/Academic Press. p. 415. ISBN 9780080497372.

21. Schultz W (2015). "Neuronal reward and decision signals: from theories to data". 853–951. doi:10.1152/physrev.00023.2014. PMC 4491543. PMID 26109341

22. Nico H. Frijda, The Emotions. Cambridge (UK): Cambridge University Press, 1986. p. 207

23. Vaidya Jadavaji Trikamji Acharya, Dalhana, Susruta. Nibandha sangraha,Susruta samhita. Varanasi: Chaukhambha Sanskrit sansthan; 2009. Chapter 1, Sootra Sthana, shloka.3; p.6.

24.Vaidya Jadavaji Trikamji Acharya, Agnivesha. Charaka Samhita.Varanasi: Chaukhambha Prakashan; 2011. Chapter 12, Sutra Sthana, shloka.11; p.80.

25. Rømer Thomsen, K; Whybrow, PC; Kringelbach, ML (2015). "Reconceptualizing anhedonia: novel perspectives on balancing the pleasure networks in the human brain". Frontiers in Behavioral Neuroscience. 9: 49. doi:10.3389/ fnbeh.2015.00049. PMC 4356228. PMID 25814941

26. Olney, JJ; Warlow, SM; Naffziger, EE; Berridge, KC (August 2018). "Current perspectives on incentive salience and applications to clinical disorders". Current Opinion in Behavioral Sciences. 22: 59–69. doi:10.1016/j.cobeha.2018.01.007. PMC 5831552. PMID 29503841.

27. Vaidya JadavajiTrikamji Acharya, Agnivesha. CharakaSamhita. Varanasi: ChaukhambhaPrakashan; 2011. Chapter 8, SootraSthana, shloka.11; p.56

28. Vaidya Jadavaji Trikamji Acharya, Agnivesha. CharakaSamhita.Varanasi: ChaukhambhaPrakashan; 2011. Chapter 8, SoothraSthana, shloka.4; p.55

29. Vaidya JadavajiTrikamji Acharya, Agnivesha. Charaka Samhita.Varanasi: Chaukhambha Prakashan; 2011. Chapter 1, SootraSthana, shloka.55; p.15

30. Vaidya JadavajiTrikamji Acharya, Agnivesha. Charaka Samhita.Varanasi: Chaukhambha Prakashan; 2011. Chapter 8, SootraSthana, shloka.11; p.56

31. Pt Hari Sadashiva Sastri paradakara,Vagbhata. Astanga Hridaya. Varanasi: Chaukhambha Sanskrit samsthan; 2010. Chapter 13, Sutra sthana, shloka.6; p.212.

32. Pt Hari Sadashiva Sastri paradakara, Arunadatta, Vagbhata. Sarvanga sundara, Astanga Hridaya. Varanasi: Chaukhambha Sanskrit samsthan; 2010. Chapter 13, Sutra sthana, shloka.6; p.212.

33. Vaidya Jadavaji Trikamji Acharya, Agnivesha. Charaka Samhita.Varanasi: Chaukhambha Prakashan; 2011. Chapter 6, Vimana Sthana, shloka.17; p.256.

34. Vaidya Jadavaji Trikamji Acharya, Agnivesha. Charaka Samhita.Varanasi: Chaukhambha Prakashan; 2011. Chapter 6, Vimana Sthana, shloka.17; p.256.

35. Vaidya Jadavaji Trikamji Acharya, Agnivesha. Charaka Samhita.Varanasi: Chaukhambha Prakashan; 2011. Chapter 6, Vimana Sthana, shloka.17; p.256.

36. Vaidya Jadavaji Trikamji Acharya, Agnivesha. Charaka Samhita.Varanasi: Chaukhambha Prakashan; 2011. Chapter 8, Vimana Sthana, shloka.87; p.275.

37. Pt Hari Sadashiva Sastri paradakara, Vagbhata. Astanga Hridaya. Varanasi: Chaukhambha Sanskrit samsthan; 2010. Chapter 13, Sutra sthana, shloka.6; p.212.

38. Govindadasa. Bhaishajya Ratnavali. Varanasi. Chaukhamba Sanskrit samsthan: Parishishta, shloka 21; p.852.

39. Bruscia KE. Defi ning Music Therapy. Ed. Enelivros. Rio de Janeiro, Brazil. 2000; 276. 26.

40. Koelsch S. Brain correlates of music-evoked emotions. Nature Reviews Neuroscience. 2014 Mar;15(3):170-180. https://doi.org/10.1038/ nrn3666

41. Music improves dopaminergic neuro transmission: demonstration based on the effect of music on blood pressure regulation https:// doi.org/10.1016/j.brainres.2004.05.018

42. Moraes MM1, Rabelo PCR2, Pinto VA2, Pires W3, Wanner SP2, Szawka RE4 et al.Auditory stimulation by exposure to melodic music increases dopamine and serotonin activities in rat forebrain areas linked to reward and motor control. PMID:29499311DOI:10.1016/j. neulet.2018.02.058

 

We use and utilize cookies and other similar technologies necessary to understand, optimize, and improve visitor's experience in our site. By continuing to use our site you agree to our Cookies, Privacy and Terms of Use Policies.